Guarding against Collateral Damage during Chromatin Transactions

Research output: Contribution to journal › Review › Research › peer-review

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Guarding against Collateral Damage during Chromatin Transactions. / Altmeyer, Matthias; Lukas, Jiri.

In: Cell, Vol. 153, No. 7, 20.06.2013, p. 1431-4.

Research output: Contribution to journal › Review › Research › peer-review

Harvard

Altmeyer, M & Lukas, J 2013, 'Guarding against Collateral Damage during Chromatin Transactions', Cell, vol. 153, no. 7, pp. 1431-4. https://doi.org/10.1016/j.cell.2013.05.044

APA

Altmeyer, M., & Lukas, J. (2013). Guarding against Collateral Damage during Chromatin Transactions. Cell, 153(7), 1431-4. https://doi.org/10.1016/j.cell.2013.05.044

Vancouver

Altmeyer M, Lukas J. Guarding against Collateral Damage during Chromatin Transactions. Cell. 2013 Jun 20;153(7):1431-4. https://doi.org/10.1016/j.cell.2013.05.044

Author

Altmeyer, Matthias ; Lukas, Jiri. / Guarding against Collateral Damage during Chromatin Transactions. In: Cell. 2013 ; Vol. 153, No. 7. pp. 1431-4.

Bibtex

@article{b6449e23ba62416c98f87dbbbc4f8242,
title = "Guarding against Collateral Damage during Chromatin Transactions",
abstract = "Signal amplifications are vital for chromatin function, yet they also bear the risk of transforming into unrestrained, self-escalating, and potentially harmful responses. Examples of inbuilt limitations are emerging, revealing how chromatin transactions are confined within physiological boundaries.",
author = "Matthias Altmeyer and Jiri Lukas",
note = "Copyright {\textcopyright} 2013 Elsevier Inc. All rights reserved.",
year = "2013",
month = jun,
day = "20",
doi = "10.1016/j.cell.2013.05.044",
language = "English",
volume = "153",
pages = "1431--4",
journal = "Cell",
issn = "0092-8674",
publisher = "Cell Press",
number = "7",

}

RIS

TY - JOUR

T1 - Guarding against Collateral Damage during Chromatin Transactions

AU - Altmeyer, Matthias

AU - Lukas, Jiri

N1 - Copyright © 2013 Elsevier Inc. All rights reserved.

PY - 2013/6/20

Y1 - 2013/6/20

N2 - Signal amplifications are vital for chromatin function, yet they also bear the risk of transforming into unrestrained, self-escalating, and potentially harmful responses. Examples of inbuilt limitations are emerging, revealing how chromatin transactions are confined within physiological boundaries.

AB - Signal amplifications are vital for chromatin function, yet they also bear the risk of transforming into unrestrained, self-escalating, and potentially harmful responses. Examples of inbuilt limitations are emerging, revealing how chromatin transactions are confined within physiological boundaries.

U2 - 10.1016/j.cell.2013.05.044

DO - 10.1016/j.cell.2013.05.044

M3 - Review

C2 - 23791174

VL - 153

SP - 1431

EP - 1434

JO - Cell

JF - Cell

SN - 0092-8674

IS - 7

ER -

ID: 47458713